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As early as1923, Kylin, a Swedish physician, described the clus-tering of cardiovascular risk factors such as hyper-tension, obesity, and gout (1). Thebinding of FOXP3 to these enhancers can also activate other transcription factors that bind toenhancer/promoter regions of genes specifically needed to construct the Treg phenotype.
In particular, they discussed two approaches to including subjects in the finalanalysis of the results of the experiment: “intention-to-treat” versus “as-treated” analysis.In the intention-to-treat analysis, all of the subjects are included in the final data analy-sis regardless of whether they completed the treatment. (2002) Diagnosis and treatmentof viral encephalitis.
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